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My first try at this

Discussion in 'Rebooting - Porn Addiction Recovery' started by Deleted Account, Jun 4, 2018.

  1. I’m on my seventh day of no porn, masturbation or orgasm. I have never gone this long without orgasming in some way since I was around 12. But now my dick feels like dead or something - I normally have hard-ons a lot of the time but right now - and for the majority of this streak - it's been nothing. I'm on Prozac so maybe that's part of it - but it's also super abnormal for me this whole situation and it's scaring me - has anyone else experienced this?
     
  2. Darth Buji

    Darth Buji Fapstronaut

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    Dude, congrats on 6 days! I remember the first time I went an entire week was pretty similar to how you described - just a general daze and almost no sexual urge. But, trust me, soon enough it'll be the complete opposite problem. You'll be jonesin' to use again, and if you conquer that, you'll be golden! Keep up the good work, Fapstronaut
     
  3. Thanks! If you don’t mind me asking what was your experience with this? Any tips?
     
  4. Here's an article from a website I find helpful. Please enjoy.

    There are several types of anti-depression drugs. Widely used are SSRIs (Selective serotonin reuptake inhibitors) and SNRIs (Serotonin-norepinephrine reuptake inhibitors). Usually, anti-depression drugs lead to excessive chronic prolactin production – a condition called Hyperprolactinemia (can best be described as a functional disorder). Excessive prolactin in the bloodstream reduces the testosterone and DHT release from the testicles, resulting in bone and muscle weakness.

    SSRIs tend to block dopamine nervous functions in the hypothalamus-pituitary axis, which are responsible for the release of oxytocin, known as the emotional and orgasm hormone. The drugs also tend to indirectly interfere with all the neurotransmitter systems and the production of various hormones, usually by overloading the liver P450 detoxification system. They also block the spinal nervous transmissions in the synapses with a flooding of serotonin through a reuptaking inhibition. The gene expressions of D2 dopamine receptors, acetylcholine receptors, and 5-HT (Serotonin) 2C receptors are also being alternated.

    SSRIs will destroy the pituitary and liver functioning, burdening your endocrine system. SSRIs not only inhibit the reuptaking of serotonin, but actually lower the natural levels of serotonin due to the destructive effects on the liver P450. SSRIs drugs usually reduce the serotonin nervous action in the neuromuscular ending receptors all over the body.
    The blocking of dopamine and acetylcholine transduction, accompanied with the high level prolactin (being dumpled into the bloodstream), disables the nervous function in the penis and seminal production, and if taken systematically, can completely desensitize the penile nerves, prostate and seminal vesicles. Furthermore, SSRIs drugs tend to block hypothalamic/adrenal – dopamine – norepinephrine conversion and oxytocin release (essential to activation of orgasmic contractions).

    SSRIs anti-depression drugs cause neuroplasticity, a term used in this particular context to describe deformation of synapses and outgrowth of serotonin neurons. This neuroplasticity is known to cause addiction and severe withdrawal symptoms. SSRIs will inhibit the reputaking of serotonin in the nervous synapses by blocking the serotonin transporter, thus allowing the serotonin neurotransmitter to continue influencing the postsynaptic neurons. However, SSRIs are not increasing neurotransmitter synthesis, on the contrary – they are cutting down the natural serotonin release from the nervous terminals, it is just that SSRIs stimulate more serotonin neurons. SSRIs antidepressants also tend to alter the relationship between dopamine and serotonin signaling in the striatum, which will receive moderate serotonergic and rich dopaminergic innervation. This can be extremely harmful and may lead to irreversible damage. Due to the inhibition of serotonin transporters, the dense striatal Dopamine Transporters may uptake serotonin into the dopamine terminals, which will destroy the dopamine nervous function responsible for the release of oxytocin, LH and FSH. Also, a weak dopaminergic function will result in heightened pituitary release of prolactin and disable sexual function completely.

    To negate the ill effects SSRIs, one must power his dopamine, cholinergic, serotonin and GABA nervous function. Dopamine is essential to the hypothalamus-pituiatry-testicular function.
     
  5. Eh? I mean I’ve taken SSRI’s in the past they inhibit my ability to orgasm but I need to take them as I have major depression, ocd and anxiety disorder. It’s impermanent but I still have to be on them for a bit to function properly. I’ve alwaus been able to get aroused on it, just not as much satisfaction from orgasming and it’s harder to orgasm, this is the first time I haven’t orgasmed for 6 days straight since before I can remember and the effects are odd.
     
  6. Despite what Big Pharma and Big Med would want you to believe, depression and anxiety aren't SSRI deficiencies. At best, they mask symptoms. At worst, they completely fuck your body's natural ability to perform many of its most needed functions through an unnatural unbalancing of hormones and neurotransmitters. But hey, that's just me. I'm probably just full of shit.

    #foodforthought
     
  7. ROCKETMAN1

    ROCKETMAN1 Fapstronaut

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    Ever been on them? I have for years; I have no ill effects. Who wrote that article and what are his credentials so I can look it up? I’ve never seen any doctor or scientist make all those claims about SSRI’s destroying certain pathways, systems, etc. Sexual dysfunction including difficulting reaching orgasm and ED are a common side effect. I majored in cognitive neuroscience and have taking psychopharmacology classes and most of what you said is not backed up by any research. Please share the source. Thanks
     

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